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Offline D1C1

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Penyakit yg diturunkan
« on: 06 May 2018, 10:52:33 PM »
Alo temen,

Misalkan kita dilahirkan di keluarga yg memiliki penyakit turunan, penyakit turunan ada banyak sekali jenisnya, jasmani dan jiwa. Contohnya, diabetes, kanker, gangguan kecemasan, dll.

Pertanyaannya, kalo kita terlahirkan di keluarga yg memiliki penyakit turunan. Jika kita suatu hari menikah dan memiliki anak, otomatis penyakit turunan ini akan turun juga ke anak kita, apakah kita berbuat karma buruk? Ada orang berkata lebih baik tdk menikah dan punya anak.

Offline seniya

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Re: Penyakit yg diturunkan
« Reply #1 on: 07 May 2018, 09:20:30 AM »
Alo temen,

Misalkan kita dilahirkan di keluarga yg memiliki penyakit turunan, penyakit turunan ada banyak sekali jenisnya, jasmani dan jiwa. Contohnya, diabetes, kanker, gangguan kecemasan, dll.

Pertanyaannya, kalo kita terlahirkan di keluarga yg memiliki penyakit turunan. Jika kita suatu hari menikah dan memiliki anak, otomatis penyakit turunan ini akan turun juga ke anak kita, apakah kita berbuat karma buruk? Ada orang berkata lebih baik tdk menikah dan punya anak.

Definisi karma itu kan udah jelas dalam Buddhis, trus tinggal dicek apakah perbuatan tsb termasuk karma dlm definisi Buddhis
« Last Edit: 07 May 2018, 09:25:09 AM by seniya »
"Holmes once said not to allow your judgement to be biased by personal qualities, and emotional qualities are antagonistic to clear reasoning."
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Offline Lex Chan

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Re: Penyakit yg diturunkan
« Reply #2 on: 09 May 2018, 12:54:39 PM »
maaf, saya menyanggah bahwa contoh2 yang disebutkan: diabetes, kanker, gangguan kecemasan termasuk dalam penyakit keturunan.
saya tidak perlu paparkan penjelasannya, silakan google. segala macam informasi yang berkaitan dengan penyakit2 itu bisa ditemukan di internet.

yang menarik bagi saya adalah penyakit yg bisa ditularkan ke anak, misalnya HIV.
nah, kalau misalnya seseorang mengidap HIV dan dia tahu bahwa dia mengidap HIV, kemudian menularkan kepada pasangan dan keturunan.
mungkin ini yang jadi pertanyaan. bukankah begitu?
“Give the world the best you have and you may get hurt. Give the world your best anyway”
-Mother Teresa-

Offline DeNova

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Re: Penyakit yg diturunkan
« Reply #3 on: 30 May 2018, 07:06:53 PM »
maaf, saya menyanggah bahwa contoh2 yang disebutkan: diabetes, kanker, gangguan kecemasan termasuk dalam penyakit keturunan.
saya tidak perlu paparkan penjelasannya, silakan google. segala macam informasi yang berkaitan dengan penyakit2 itu bisa ditemukan di internet.

yang menarik bagi saya adalah penyakit yg bisa ditularkan ke anak, misalnya HIV.
nah, kalau misalnya seseorang mengidap HIV dan dia tahu bahwa dia mengidap HIV, kemudian menularkan kepada pasangan dan keturunan.
mungkin ini yang jadi pertanyaan. bukankah begitu?
Orang tua punya HIV anak bisa negatif, atau pasangan punya HIV belom tentu pasangannya kena... Daripasda HIV yg mungkin penularannya sebenernya bisa dicegah, LUPUS adalah yg paling kontroversial

Offline kullatiro

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Innate resistance to HIV
« Reply #4 on: 03 June 2018, 11:40:06 PM »
Innate resistance to HIV

A small proportion of humans show partial or apparently complete inborn resistance to HIV, the virus that causes AIDS.[1] The main mechanism is a mutation of the gene encoding CCR5, which acts as a co-receptor for HIV. It is estimated that the proportion of people with some form of resistance to HIV is under 1%.[2][3][4]


In 1994, Stephen Crohn became the first person discovered to be completely resistant to HIV in all tests performed.[5][6] In early 2000, researchers discovered a small group of sex workers in Nairobi, Kenya who were estimated to have sexual contact with 60 to 70 HIV positive clients a year without signs of infection.[7] Researchers from Public Health Agency of Canada have identified 15 proteins unique to those virus-free sex workers.[8] Later, however some sex workers were discovered to have contracted the virus, leading Oxford University researcher Sarah Rowland-Jones to believe continual exposure is a requirement for maintaining immunity.[9]


CCR5 deletion

C-C chemokine receptor type 5, also known as CCR5 or CD195, is a protein on the surface of white blood cells that is involved in the immune system as it acts as a receptor for chemokines. This is the process by which T cells are attracted to specific tissue and organ targets. Many strains of HIV use CCR5 as a co-receptor to enter and infect host cells. A few individuals carry a mutation known as CCR5-Δ32 in the CCR5 gene, protecting them against these strains of HIV.


In humans, the CCR5 gene that encodes the CCR5 protein is located on the short (p) arm at position 21 on chromosome 3. Certain populations have inherited the Delta 32 mutation resulting in the genetic deletion of a portion of the CCR5 gene. Homozygous carriers of this mutation are resistant to M-tropic strains of HIV-1 infection.[10][11][12][13][14][15]



https://en.m.wikipedia.org/wiki/Innate_resistance_to_HIV
« Last Edit: 03 June 2018, 11:41:46 PM by kullatiro »

Offline kullatiro

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Biologists discover why 10% of Europeans are safe from HIV infection
« Reply #5 on: 03 June 2018, 11:46:53 PM »
Biologists discover why 10% of Europeans are safe from HIV infection

LIVERPOOL, UK - 9 March 2005: Biologists at the University of Liverpool have discovered how the plagues of the Middle Ages have made around 10% of Europeans resistant to HIV.
Scientists have known for some time that these individuals carry a genetic mutation (known as CCR5-Ä32) that prevents the virus from entering the cells of the immune system but have been unable to account for the high levels of the gene in Scandinavia and relatively low levels in areas bordering the Mediterranean.

They have also been puzzled by the fact that HIV emerged only recently and could not have played a role in raising the frequency of the mutation to the high levels found in some Europeans today.

Professor Christopher Duncan and Dr Susan Scott from the University's School of Biological Sciences, whose research is published in the March edition of Journal of Medical Genetics, attribute the frequency of the CCR5-Ä32 mutation to its protection from another deadly viral disease, acting over a sustained period in bygone historic times.

Some scientists have suggested this disease could have been smallpox or even bubonic plague but bubonic plague is a bacterial disease rather than a virus and is not blocked by the CCR5-Ä32 mutation.

Professor Duncan commented: "The fact that the CCR5-Ä32 mutation is restricted to Europe suggests that the plagues of the Middle Ages played a big part in raising the frequency of the mutation. These plagues were also confined to Europe, persisted for more than 300 years and had a 100% case mortality."

Around 1900, historians spread the idea that the plagues of Europe were not a directly infectious disease but were outbreaks of bubonic plague, overturning an accepted belief that had stood for 550 years. Professor Duncan and Dr Scott illustrated in their book, Return of the Black Death (2004, Wiley), that this idea was incorrect and the plagues of Europe (1347-1660) were in fact a continuing series of epidemics of a lethal, viral, haemorrhagic fever that used the CCR5 as an entry port into the immune system. Using computer modeling, they demonstrated how this disease provided the selection pressure that forced up the frequency of the mutation from 1 in 20,000 at the time of the Black Death to values today of 1 in 10.

Lethal, viral haemorrhagic fevers were recorded in the Nile valley from 1500 BC and were followed by the plagues of Mesopotamia (700-450BC), the plague of Athens (430BC), the plague of Justinian (AD541-700) and the plagues of the early Islamic empire (AD627-744). These continuing epidemics slowly raised the frequency from the original single mutation to about 1 in 20,000 in the 14th century simply by conferring protection from an otherwise certain death.

Professor Duncan added: "Haemorrhagic plague did not disappear after the Great Plague of London in 1665-66 but continued in Sweden, Copenhagen, Russia, Poland and Hungary until 1800. This maintenance of haemorrhagic plague provided continuing selection pressure on the CCR5-Ä32 mutation and explains why it occurs today at its highest frequency in Scandinavia and Russia."



https://www.eurekalert.org/pub_releases/2005-03/uol-bdw031005.php

Offline kullatiro

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Timothy Ray brown first patien cure from HIV/Aids
« Reply #6 on: 03 June 2018, 11:51:53 PM »
Timothy Ray Brown

Timothy Ray Brown (born 1966) is an American considered to be the first person cured of HIV/AIDS.[1][2] Brown was diagnosed with HIV in 1995 while studying in Berlin, Germany, giving him the nickname The Berlin Patient.[3]

https://en.m.wikipedia.org/wiki/Timothy_Ray_Brown

Offline kullatiro

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2017 six patient hiv/aids been cure
« Reply #7 on: 04 June 2018, 08:39:35 AM »
As of 2017, six more people also appear to have been cleared of HIV after getting graft-versus-host disease; only one of them had received CCR5 mutant stem cells, so it appears that when a transplant recipient has graft-versus-host disease the transplanted cells may kill off the host's HIV-infected immune cells.[11]

https://en.m.wikipedia.org/wiki/Timothy_Ray_Brown

 

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